#460: Most Impactful Pod Moments: Hidden Cancer Markers, Radical Longevity Experiments, EMFs Exposed (and the Mast Cell Dilemma) With Nathalie Niddam & Guests

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Welcome to Longevity. I'm your host, Natalie Nidham. I'm a nutritionist, a human potential, and epigenetic coach, and I created this podcast to bring you the latest ways to take control of your health and longevity. We cover it all, from new technology and ancestral health practices to personalized interventions, and a very special interest of mine, peptides and bioregulators. Enjoy the show.
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Welcome back to the show, folks. I'm Natalie Nidham, your host. Today's episode is a little bit different. Instead of an interview or a solo-sode, what we're doing is we're looking back at some of the amazing episodes from the past quarter.
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So we've done this before and people really enjoyed it because either it reminds you of an episode that you really loved, or you might just hear something here from an episode that you might've missed. Not to say that we didn't have amazing guests every single episode, but these were some of the episodes that just really stood out to me and the teams.
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So hopefully you agree. If there's episodes that you love better, then make sure you let us know either in comments on Instagram, on my new account, at longevitywithnat, or maybe on YouTube there's comments. And I think Spotify also allows you to leave comments. So let us know in the comments what you love and we will take it all into consideration for next time.
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In the meantime, new Instagram. So please, please make sure to follow my new Instagram account at longevitywithnat. And if you're loving this show or there's episodes that you especially love, please make sure...
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to leave us a review and also to share the episodes with anybody in your world who you think would love them as well. All right, here we go. Let's talk about these episodes that really moved the needle on longevity in the conversations. We've selected snippets about how to know if your body's terrain could be hospitable for cancer, how to calm the nervous system when dealing with mast cell issues you probably didn't know you had in the first place, and
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As you know, mast cell issues do tend to creep up as we age folks. Now, I wanted to highlight once again, the critical nature of Lyme misdiagnoses that could even look like Alzheimer's. And while I know this audience is likely familiar with EMFs, I wanted to pull in a clip about how to choose the best products for EMF protection.
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Lastly, the long COVID conversation continues in the health world and Dr. Robin Rose breaks down ways to mitigate the negative effects. While all of these things can sound scary on their own, these experts don't just bring their scare tactics. The reason we chose them and the reason we love these conversations is that they also talk about what you can do to help. So here we go.
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In episode number 436, Dr. Naysha Winters discusses the essential lab markers that she looks at to indicate a disease state that might, might, doesn't always, but it might signal cancer. You're going to want to write these down. So you need a pen and paper for this one, for sure. Newsflash.
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I have officially reached the point where I don't get excited by skincare that promises to erase 10 years overnight. Here's the thing. We all know this. If it sounds too good to be true, well, my bathroom cabinet and probably yours has enough evidence already that it's just not a thing. Now, this is one of the reasons why Vita Zero Age Exosomes from Vitaly caught my attention.
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Instead of focusing on covering up aging, it's designed around the biology of how skin communicates and repairs itself. What's unique here is what they call regenerative stacking. You've got zero age mesenchymal stem cell exosomes acting as the messengers paired with GHKCU to support rebuilding the skin structure. Then they layer in niacinamide and glutathione. So you're also supporting barrier and oxidative stress.
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I love that it fits with the same longevity mindset that you and I use everywhere else, supporting healthier tissue, healthier cells, and healthier skin over the long haul. Now, if you'd like to check it out for yourself, head over to Vitali Skin Care and make sure to use code NAT20 for 20% off your order.
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The term longevity is everywhere these days, and I do talk about it a lot. But here's something that we don't discuss nearly enough. Healthy aging isn't just about adding years to your life. It's really about making sure that you remain vital right until the end when you're done. And to do that, we want to make sure that your cells are still doing their job along the way.
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This is one reason why Mito Blue from Wizard Sciences caught my attention. It combines pharmaceutical grade methylene blue with ingredients that support some of the most important longevity pathways that we know about today. You've got NMN to support healthy NAD levels, PQQ to help promote the creation of new mitochondria,
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fisetin to support cellular cleanup, and apigenin for healthy aging and mental clarity. What makes it unique is that it's focused on both sides of the equation, helping clear out what isn't serving you while supporting the systems that help your cells to thrive. Healthy mitochondria, healthy energy, healthy aging.
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To learn more, check out wizardsciences.com and make sure to use code NAT15 for 15% off at checkout. Are there three to five data points that immediately tell you,
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Okay, here's what we've got going on here. And, you know, I think you've already talked about one of them just now so beautifully. Yeah. Well, this is simple. When I was in private practice for 17 years, even though I tried like hell not to treat cancer patients, they still kept finding me. All right. And so people with cancer, they still kept finding me. But what I did do every year, twice a year in Durango, Colorado, there was this statewide process called the 9R Health Fair.
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So for about 60 bucks twice a year, people from all over the state could go to all these little testing stations and get their labs drawn. And they got everything from a CBC and a C and this is back all the way through until about 2000. It still happens by the way, but imagine 60 bucks today as you can get a lot of tests for that. But back then those same out of pocket would have cost you several hundred, if not a thousand dollars.
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And we're talking CBC with differential, C and P, vitamin D level, C-reactive protein, a TSH for men, a PSA, and a hemoglobin A1C. That's where that test ran for about 60 bucks twice a year. So I trained my clients to get those tests done twice a year. And I set up basically little tests.
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meet and greets to review their labs twice a year regarding that. That might be the only time I ever saw anybody. But what I learned that in 17 years of doing that in private practice was I saw a lot of trends, a lot of patterns, and we got to catch things way early and turn them around in the harbor before it became a problem.
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Right. Beautiful. And so the biggest levers, the biggest data points that I learned were. And in fact, this wasn't initially done on that test, but the CBC, this is even today you could run for five bucks. Right. It's like nothing, nothing. And yet this tells me everything I need to know about your immune system.
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Right. So this is looking at lymphocytes, neutrophils, the ratio between them. These are white blood cells. So these are immune markers and they're not the fancy thing. They're not your TNF alpha or whatever, but you don't even, I'm guessing you don't even have to go that deep initially to see is the footprint there. Right. Right. In fact, for Oncology Nutrition Institute, I still have, it's several years old now and it's still as relevant as ever, but I have a 90 minute course that I think just sells online for like 10 bucks or 15 bucks.
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of this 90 minute course on just understanding the CBC. Like it is such a gold. That would be gold people. If you understand your CBC that, I mean, I've been deep in my CBC for the last two months. It's amazing the amount of information you can pull from there. Yeah. And we want accessibility, right? And so when you can spend $5 and $6 on a draw fee and $5 on the test itself, it's to me worth everyone's investing that 10 to 15 bucks
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to get this test that can tell you so much because this could have had a lot of things off in the past. In fact, your listeners could go and take a look on PubMed and just simply type in neutrophil to lymphocyte ratio. Just type that in and look at the tens of thousands of papers on this. And one of the most significant papers is that that simple little ratio is the most prognostic marker for all cause mortality.
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whether that's cancer or not. So when we're on the sexy, I mean, we're on the longevity show for Pete's sake, this is one of your most profound longevity markers. Beautiful. Okay. So CBC, what else was HBA1C? For me, my three are, or my five are very, and these are what I've figured out over my years, but the CBC with DIF, the CMP, which is your metabolic panel, which shows me your organ function, your electrolyte function, and just kind of
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How's your body doing? It's kind of like a very, it's like taking your car to the mechanic and having them run the electric, you know, just to like see what's going on. And then the three, my patients have coined this as the trifecta. Okay. And this is a collection because any one of them by themselves are very, very informative, but together they are, for me in the oncology space, my everything. Okay. So the C-reactive protein used to just be a marker of general inflammation. Okay.
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And it still is. It's still a very powerful tool to understand your inflammatory processes, which we know drives things like mental illness, autoimmune conditions, cancering process, cardiovascular disease. All of the diseases that plague us today are very inflammatory driven. So there's that. The next one used to be part, by the way,
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of every CMP, of every metabolic panel until about 20 years ago, someone behind a desk decided that was not cost-effective and pulled it, is the lactase dehydrogenase. And frankly, folks, this is your best test for your mitochondrial function without spending hundreds, if not thousands of dollars on testing your mitochondrial function.
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And so the lactase dehydrogenase is a way that it's an average of five different enzymes of the body. So it kind of gives you an overall score. And you can even get an LD isoenzyme to break it down to understand which tissues are most perturbed and how to deal with it.
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LD1 related to your blood cells, LD4 to your lung, LD3 to your bone. Like you can even know which tissues of your body are most disrupted with that. If you run an isoenzyme, which is a little bit more expensive, but not much. And then the third, which also used to be part of an old CMP,
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is also another powerful standalone test. We use it a lot in autoimmunity, which is the sedimentation rate or ESR. Okay. This little bugger is about basically how fast are your cells falling out of solution? So how thick and sticky is your blood and how inflamed and fibrinous is your blood? Because that leads to poor oxygenation, that leads to hyperinflammation, that leads to hypercoagulation, that leads to a lot of issues, right? It makes sense.
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fluid in the system. So when any one of those are elevated, that's a problem, right? When two of the three are elevated, that's something that's worth investigating, right? A single one could be like, oh, I just stubbed my toe really hard before going in for my, you know, blood work. Or I did a crazy workout yesterday. Crazy workout when your LDH looks scare scare, right? So those types of things. But if all three are high, I'm looking for cancer.
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And I don't tell people this to scare them. I tell people this because it means you can do something about it before it's a problem. And so for me, the trifecta is far more telling than our cancer markers. So like our CEA, our CA-125, our CA-2729, those are markers for things like colorectal cancer, ovarian cancer, breast cancer. Those are not specific or sensitive enough. In fact, CEA can be high if you just had dental work.
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Right. Or a C125 can be high if you just had sex. So these are things that you've got to look at more data points. The other side of it is we look a lot on imaging to guide us. And so here's what I'll tell you and your listeners. If I'm looking at someone's labs and they just rang the bell in their finishing chemo or radiation and their markers, their cancer markers are perfect...
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And their imaging is perfect, but their trifecta, all three more elevated. I am frankly squeezing my sphincters like you can't even imagine because I know after 30, almost 35 years of looking at this type of data, and it's unfortunately very, very accurate.
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Or fortunately, for me, fortunately, for some patients, they freak out, and this is what prevents them from wanting to look under the hood. But if I see a trifecta that's all elevated following a clean scan, clean labs, I know that this person will be cancering again within days, weeks, or months, and pretty aggressively.
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So the process hasn't stopped. It has not stopped. In fact, it's just now we've pushed back all the daughter cells, which are the active proliferating cancer cells. But what that shows me is we've now woken up mama stem cells and they are more difficult to find, more difficult to treat. And they're a different animal than the original cancer source. And so these are the things that I'm looking at this process before treatment for prevention.
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during treatment and after. And we're monitoring this closely because we can watch these trends. So if I have a patient who still has stuff on their images that still have elevated cancer markers, yet their trifecta is perfecta, they can live a very long vital life with cancer because their terrain is in the driver's seat and not the tumor. That's interesting. And then could, but then also could those,
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more obvious markers of cancer start to go down over time, essentially, because the terrain is no longer hospitable to it. Yes, exactly. In episode number 434, Dr. Sandy Kaufman breaks down mast cells, how common mast cell issues have become and ways that people can mitigate the effects. She also points out the shocking connection between
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estrogen, and mast cells. So for women going through menopause, this is a big one, but there's something in here for you if you're a guy or if you're a woman. Let's dive into one of your recent kind of pet projects, mast cells and immune aging. All right. So I had to give you some juice here, obviously.
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So when you look at aging through an immune lens, what changes first? Surveillance, tolerance, inflammatory tone or recovery after insults? Or it depends on the person. Oh my God, that is such a loaded. I'd have to go with E all of the above. Exactly. All of the above and it depends. It does. It's all of the above, right? Your immune system fails so that you get more infections. You can't fight things off. Things that are in remission come out.
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So tuberculosis is why people get shingles, right? You don't really respond well to vaccines anymore. All the things that are good go down, all the things that are bad go up. Right, yeah, no, that makes total sense. Unfortunately, that's just the case of it. All right, go ahead. Right, so mast cells are really interesting because they go up.
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which means by default that they are the villains in our story. When you do skin biopsies, because it's easier to do skin than anything else, you see a 40 to 50 to even 60% increase over time.
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Really? Yes. In the presence of mast cells as we age? That's correct. Okay. All right. What do mast cells do? Mast cells are packed. They're called mastzallen as named by Paul early in the 1800s because under a microscope, they looked stuffed. They looked well-fed because they have all these granules in there.
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Right. So there's a big nucleus and then a ton of granules. And when they granulate, they sort of release these things and they go everywhere. And it's these molecules that cause problems everywhere. And they could release like 200 or so different types of molecules, including but not limited to because there's a whole lot of them. The one everyone recognizes is histamine.
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which causes all of our immune allergy type stuff. Proteases, chymases, MMPs, all your interleukins, your tumor necrosis factor, again, a gazillion things, right? It's a lot of inflammatory molecules. Tons of inflammatory molecules. And there's some stuff that's beneficial. Don't get me wrong. They're not all evil. But in general, they're extremely inflammatory. And they dissolve things. And my real pet peeve here is the proteases.
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So proteases, by definition, is dissolving of proteins. Yeah, it sounds like a digestive thing. It is. It is. So they release chymases and tryptases, and they literally...
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dissolve stuff. And they activate your MMPs, and that dissolves collagen one and three. So you are dissolving yourself as these mast cells degranulate. And as you get older and you have more, they degranulate more and more and more frequently. And as you have areas of pathology, they accumulate and they degranulate. And you are literally dissolving yourself as you get older. And this is one of the big reasons that, so if you have coronary disease,
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They accumulate in areas of atheroma and you piss off your mast cells. You just had a heart attack. They they aggregate in your intima of your your abdominal wall. So if you have a triple A, an abdominal aneurysm, it causes it to rupture because it's dissolving your tissue.
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That's amazing. So how do we stop that as we age? Because this is an aging problem. So this is an allergy problem. Yeah. This is an aging problem. This is not just this person, that person. This is an everybody problem. Okay. So you can either, the only thing that you can actually do individually is to block histamine. And people are big on that, right? Taking H1 or H2 blockers. Or what I like to do is stabilize the entire mast cell.
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And what I have done is I went through, so mast cells degranulate, there's probably 20, 25-ish different receptors on a mast cell causing it to degranulate. That's incredible. So depending on the receptor and the antigen, different types of things will get released, right? The two main receptors is the IgE,
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excuse me, and then the MRX, and then there's a bunch of other ones too. For example, there's a receptor for corticotropin releasing hormone, so you can actually be allergic to stress.
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Well, you know what? That's so interesting you say that because lately, if I get super stressed, I mean, I've had a thing going on lately. If I get really, really stressed, I've noticed that I can get itchy. Yeah. And I'm like, okay, that's ridiculous. You're being hypochondriac. But that now is starting to make sense. It's destabilizing those mast cells.
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Your CRH is going to a receptor, you have receptor one and two on your mast cell, and it causes it to degranulate. So you're releasing histamine and other inflammatory products, and it's causing you to quote unquote, be allergic to stress. So that is absolutely not crazy.
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That's not a good longevity strategy. No, it's not. You can respond to other things too, like changes in temperature can, can, can, can make up mast cells. Right. Yeah. Um, oxidized LDLs can do it. Also medications can do it, you know, like, like it's the number of things that can set off your mast cells is, is, is astronomical. So as a consequence of that, how, how do we defeat this? And so I'm big now into mast cell stabilization. Makes sense. Right.
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So there's probably a list. I'm about to release an e-book on Amazon. My graphic designer is beautifying it for me at the moment. Yeah. We want it to be pretty. Right now it's pretty ugly. Yeah, it'll be pretty. And it has probably about 30 over-the-counter agents that are mass cell stabilizers. And it tells you exactly which receptors it stabilizes and how well it does it.
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Took me a very long time to figure that out. So if you want natural agents, those are in there. And it's stuff like PUFAs and butyrate and PEA and a whole lot of other things. Quercetin, of course, is in there, but it's not as strong as people think that it is. Yeah. So how do people know, how can people figure out which one they need to? Well, so the question is going to be, right, is if you have standard allergies, you're going to look for things that say IgE.
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Right. Right. Right. If you don't have standard allergies and you still get that pseudo histamine itch, like, for example, a lot of white women like I have it had it like micrographia. If you scratch yourself and you light up like red, you can look attached to you.
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Yeah. That's not an IGE. That's an MRS. Right. Right. Right. So you're going to use a different stabilizer for that than you would for the IGE. Or you could use combos. Right. So honestly, all of mine are stabilized. I use like, you know, there's even a cannabinoid receptor. So PEA isn't actually a cannabinoid, but it
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it acts sort of as a cannabinoid because it prevents a true cannabinoid from being metabolized. Anyway, so it affects your cannabinoid receptors. So PEA is a stabilizer. And I love that one because it's also, it does other things like it's a,
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It's a PARP alpha agonist. It does a whole lot of other things. So one of my new favorite things is PEA. Yeah, it's a darling molecule that gets talked. It comes up a lot. Yeah, it does a lot of things. It's still figuring out where, like it seems to fit a lot of different roles.
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Yeah, it's pretty small. It's considered like a stress molecule as well. So when you are stressed, your body makes more of it. It's a sort of self-protectant, which is kind of cool. That's cool. If you take it as a supplement, the bioavailability is terrible, so it has to be micronized, just as a side note. Okay. Pro tip, people. Pro tip. In terms of medications, chromolin, of course, is the most famous mast cell stabilizer. We use that as an inhaler for asthma. Okay, great.
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But for longevity, the problem is the half-life is very short, and it doesn't really get systemically activated. So if you just have breathing problems, kremlin's fine. But for longevity purposes, you needed something that's oral. There are a variety that you can theoretically get, but since the blockade and all the taxes and stuff, you can't. So the only real one that you can get at the moment is catafatin. You mentioned that to me recently. Yeah, it's an H1...
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and mast cell stabilizer. It will make you sleepy. It's a nighttime. It's also, it's a leukotriene agonist.
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So it also prevents a whole lot of other things. And it's a phosphodiesterase inhibitor. So you get increased blood supply. And it helps you sleep. Some people think it makes you too groggy. So I take a standard dose if you have like a mast cell true diagnosed problem. It's two milligrams a day. One milligram twice a day rather.
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Talk to your doctor before you can run out and buy it. I just need to say this. Yeah, I know. For longevity purposes, I found that like a half a milligram at night is quite nice. Let's talk about where hormones fit into all this. Like, why do... Because we're seeing this, right? I think we're seeing as women go through perimenopause and menopause, they're getting these symptoms. And all of a sudden, somebody's saying to them, you have a mast cell issue. And they're like, what are you talking about? I don't have a...
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I'd be one of those people. I don't have a mast cell issue. I've never had a mast cell issue. But it seems that the hormonal shifts really kind of pulls back the curtain and brings out the reactivity. Yeah. Well, I think that what we don't realize, because, you know, there's so many things to think about in terms of longevity, but estrogen receptors are everywhere. Right.
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everywhere. We think about the standard nuclear alpha and beta receptors, right? But there's membrane receptors, there's misreceptors, they're everywhere. They're on every cell type that we have, right? And your mitochondria, they're everywhere. And so as soon as your estrogen goes down and you give more E1 than E2, which happens after menopause, it fails, right?
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So we're looking at so many problems, right? So then the question is, is how do you fix all of those problems? So I am trying at the moment, we started talking about this, is try to dissect which receptor types are where, what they do, and what do we use to fix it?
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Yeah. Right. Right. Now we use the old like throw estrogen on everything maneuver. Estrogen being usually like an E2 variety. Right. Some people use E3. I'm beginning to think that E4, which is a placental created version. So you only ever make it when you are pregnant. But it has a lot of the it's orally available. The bioavailability or the half life is like 30 some hours, which is amazing. Mm hmm.
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apparently has a neutral effect on breast cancer issues. So that's alpha table. On the other hand, it doesn't really change your cholesterol patterns. So there's going to be, right, there's going to be pros and cons to every type of selection, right? Yeah, yeah. Anyway, I'm working through that at the moment. So there will be a very detailed algorithm or process.
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graph or box or something. Something's coming. Then that, da-da-da-da-da-da-da-da-da-da-da. I think people do have made this connection, but it's part of the reason why if we want to live a longer, healthier life, and particularly as women,
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If our estrogen is going to fall off, is going to disappear on us, we're going to have to figure out a way to somehow restore that level to some level so that the whole system doesn't fall apart behind it. Like it's not. And, you know, when we're talking about estrogen going away, we're talking about your sleep tanks, your brain fog increases, your anxiety goes through the roof, your skin changes.
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disintegrates and you get you get more flares you might become intolerant to exercise like you know how do we and part of it is in I guess in your world it's how do I
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take this apart and figure out what's due to estrogen, what's due to something else. Are the mast cells being driven to the loss of estrogen? It sounds insanely complex and yet you seem to be able to develop these roadmaps that give you these big mind maps, like if yes, then this and if no, then oh, we're going to go in that direction, which I guess is part of the genius, but then you turn it into a book somehow.
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Well, we'll get there. Estrogen is not in the book right now. The estrogen is... Well, now it's your pet project after the rest. It is, in fact, my pet project, and it will be a book, and I am working on it. But because it is really interesting how one thing does lead to another, right? Because there are estrogen receptors on mast cells as well as 20 other receptors. Yeah.
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And what leads to what, what do you need to replace, what do you not replace? How do you minimize risk? Like I am retrospectively very upset about the study in 2002, the WHI that said breast cancer,
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goes up and everyone stopped taking hormones. Well, what they failed to tell you, of course, is that they started it when these women were in their late 60s and 70s. And smoking. You know, a thousand of other variables. And of course they had issues, right? So now an entire generation of women is sort of hosed because they didn't store hormones on time. Well, that's ridiculous. Luckily, I was dumb enough. I didn't know about that. I just started my own hormone replacement when I felt like it.
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And, you know, so I've sort of lucked into it. But the question is, is what is right? What is wrong? Do we need to? The other thing that no one talks about is cycling of hormones. I actually think that's going to be important. I think that we need to sort of match a normal physiologic cycle because
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Anything that has a receptor gets up, this receptor gets up and down regulated over time with stimulation, right? Interesting. So I'm wondering, and I'm just hypothesizing here, so some experts can be like, she's full of shit. And the answer is, I might be full of shit. I don't know. But it's possible that long-term estrogen causing problems may have to do with chronic upregulation of receptors. And if we cycle it and that doesn't happen, we may alleviate some of the badness.
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In episode number 450, Nicole Bell tells us the alarming statistics surrounding Lyme and other co-infections. This episode was one that really stopped me in my tracks. This information has got to get out there and become more widely known. You'll get it when you hear her speak, but if you miss this episode, then I highly, highly encourage you or strongly encourage you, I guess is the better word, to check it out.
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Let's talk a little bit about Lyme disease. Most people think it's, you know, it's the bullseye rash. All you need is a course of antibiotics. Yes. You'll be fine. So how incomplete, what are we leaving out here? How incomplete is that picture? Yeah, it's very incomplete. And I think if you look at the research and you look at the data,
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you know, that typical bullseye rash only occurs in about 14% of patients. No way. 14, 1-4. 14, not even 40. No, 14, 1-4. And, you know, and even in that case, even in the best possible, most characteristic rash, doctors will still misdiagnose the rash about 20% of the time, right? So that's like your best case scenario with rash presentation. Another about 60% of patients, you know,
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get a rash, but it's different. It's diffuse. It's often even more, you know, that gets misdiagnosed over 30% of the time. And so it's really complicated and it might be multiple rashes. It might be people, we often hear, well, people told me it was a spider bite, right? Or something of that sort. And then about, you know, 20 to 30% of patients get no rash at all.
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right? And so it's a very, if you combine all of the statistics, this is analysis that we did when I did the state of Lyme disease research for the Center for Lyme Action. It shows that with rash presentation, you're over, there's the likelihood of getting misdiagnosed is over 50%. It was 52%. And so that's not good odds, right? That's essentially as accurate as a coin flip. Given what you're telling me, like,
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It would be an underestimate. Yes. Well, because there's, as we all know in the Lyme community, right, they say, for example, there's 476,000 new patients diagnosed with Lyme every year. But that's diagnosed in the insurance system. There's one, a lot of people that are diagnosed outside of insurance claims because a lot of patients.
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you know, carriers will not cover, especially in non-endemic areas. And then also a lot of people just don't get properly diagnosed, right? They get tested and they test negative. They don't know that they have Lyme disease and they attribute it to other things. They get diagnoses like, in my husband's case, Alzheimer's. You have carditis, you have mental health disorders. I think that's one of the things that,
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really needs an awareness is that, you know, there's linkages to neurological, neuropsychiatric conditions. You know, Bartonella is linked to schizophrenia and Panspandas and other types of really serious neuropsychiatric conditions. There's, you know, my favorite is chronic fatigue syndrome. I'm like, that's not a diagnosis. That's naming your symptom. And so- A hundred percent. Yeah. So these are the things that we believe-
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the situation and the prevalence is way higher than what's out there. And a lot of that is driven by inaccurate understanding in the traditional medical community. And then also, as I said, the poor diagnostics that are out there. Right. So speaking of the poor diagnosis, why are these tick-borne infections so uniquely good at evading detection inside the human body? What makes them so darn hard to find? They're there. Yeah.
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They are, but they are sneaky. And when you really understand and you look at these pathogens, if you just look at Borrelia, you know, Borrelia, which is the bacteria causing Lyme disease, it's really a master at manipulating the immune system. It physically evades the immune system, like literally swims faster than any of your immune cells. So it literally can outrun your immune system. It actively evades.
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changes your cytokine expression, right? So it'll upregulate things that promote tolerance and kind of downregulate things that will cause an immune reaction and it can change your T cell and B cell behavior. It can you know, burrow into immune privileged sites like the brain and the joints where your immune system really can't respond properly.
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And it also but the most important thing, I think, is it likes to be in tissues. Right. So if you're thinking about a blood based test, it's not going to find it. It's not going to find it. And this is the analogy that our founder at Galaxy often uses is like, look, if you if you dip a bucket into the ocean and you pick up the bucket and you look at it and you say, nope, there's no jellyfish in the bucket.
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does that mean there's no jellyfish in the ocean? Like, no, it just means that you haven't been able to capture it in your bucket or the analogy in diagnostics is your test tube, right? And so since Lyme doesn't like to be in the blood,
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it's very difficult to do what is known as direct detection, where you're actually looking for evidence of the pathogen itself. And as a result, we've used antibodies as a marker for, you know, okay, have you seen this pathogen? Are you having a reaction to this pathogen? But there's many, many ways that antibody detection can misdiagnose.
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Well, you just said it can outrun the immune system. If it outruns the immune system, you're not producing antibodies. So that is wild. And you can't go around biopsying.
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all of a person's tissues, I'd like to take a quick second to talk about something I'm genuinely excited about. And that's the Wolverine formula from Level Up Health. You guys know how much I talk about peptides and bioregulators on this show, the science of signaling your body to repair, regenerate, and function the way it did when you were younger. Well, the Wolverine formula is
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in a single product. It's designed around recovery and tissue repair, containing peptides that support musculoskeletal healing, reduce inflammation, and help your body bounce back faster after tough workouts. Whether that's from exercise, injury, or just the wear and tear of daily life, think of it as giving your body's repair mechanism a serious upgrade.
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What I appreciate about Level Up is that they don't cut corners and there's no fairy dusting here. The formulation is thoughtful, the quality is there, and it's built for people who actually take this stuff seriously, which if you're listening to this show, probably is you. So if you're in a heavy training cycle or you're recovering from an injury, you're
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Two or three cycles of this formula would probably go a long way to supporting speedier healing. If you want to check it out for yourself, head on over to leveluphhealth.com and check out the Wolverine formula and make sure to use code NAT for a 15% off discount at checkout. Your body does it all for you. This is one way to give a little back. Once again, that's leveluphhealth.com and make sure you use code NAT for 15% off.
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We've talked about how these pathogens impact cognition, right? But what about mitochondrial health and the nervous system over time? Yeah, I mean, there's so absolutely. And I'm not the best person to talk about some of that, but I think we see it regularly in our patients.
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And not just the pathogen itself, but also the therapies. So oftentimes in the treatments, people can be on long-term antibiotics, right? And of course, that's going to cause mitochondrial dysfunction because it's essentially, you know, causing mitochondrial dysregulation and impairment. Yeah.
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it is absolutely something that is linked and causal. And there's definitely providers that are more skilled about talking about that than I am. But absolutely, the longevity aspect and the impact that it can have, whether it's severe, like where my husband's got or, you know, case got, or whether it's something that, you know, again, if we had caught it 10 years earlier and it was just kind of a minor issue that needed to be rebalanced, those are key. And I think there's a lot of,
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ways in a lot of providers that we work with that are using herbal therapies and immune you know, modulation to help restore the balance so that you can fight off the pathogens. Because again, a lot of people get exposed. It's not like everybody that gets a tick and everybody that gets Lyme disease is going to descend into chronic illness. And so the key is, is the people that are not, why is that? And how can we mirror that in a broader population base so that people don't descend into these critical conditions?
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Do you think we're even getting close to a major paradigm shift in medicine where, like, are we getting there? Do you think it's becoming even part of the landscape where infections become part of the conversation around these things like autoimmune disease, psychiatric illness, cognitive decline? Like, are you seeing any glimmers of hope here? I am. I'm seeing quite a bit. I mean, I think one...
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You know, the functional medicine community is really beginning to understand and grab on to this concept of the fact that, you know, infections are a big root cause of illness and it needs to be on the differential. It's not the answer to everything, right? There's a lot of causes of immune dysregulation and chronic disease, but it's something that should be considered.
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I think that going back to the discussion about Alzheimer's, the pathogen and the pathobiome role in Alzheimer's is starting to get traction. And the Alzheimer's Pathobiome Initiative, which was kind of thought to be crazy outliers, is starting to get in the conversation for large grant funding and for research and getting...
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mentioned in major Alzheimer's journals, which would not have happened five years ago. And so, and they're getting the publications out there to show that this is a key root cause that needs more funding and research. And then I think there's companies like us, you know, Galaxy, we are currently, most of our users are in the functional medicine community at this point, but we're working to make our technology mainstream.
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Like our Lyme urine antigen test, we are working to get that through the FDA and to get it in the hands of primary care physicians so that when you go to your doctor after you've been exposed to a tick or a known exposure or you just have the summer flu and you want to know what's going on.
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you can get a test that will actually tell you, you know, weeks before an antibody test would. And then again, antibody tests miss people even in regular environments. And so getting that urine antigen test in the hands of mainstream providers with much better diagnostic capabilities is our mission. And we also have co-infection panels looking for Bartonella and Babesia and relapsing fever Borrelia. And
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And because again, a lot of pathogens that you need to consider, it's not just about Lyme disease. And so, and there's a lot of people in the community that are really working to drive change. So I've never been more hopeful. I think the government's starting to pay attention. The CDC has updated their website and they're working with advocacy to update other parts of their website to reflect more accurate, you know, assessments of where we are with these pathogens.
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The Department of Health and Human Services held a roundtable in December of 2025 to acknowledge that tick-borne disease is a major cause of chronic conditions in the United States. And that it needs to be a focus and that it will be a focus area for HHS. And so change is happening. But change happens slowly in the medical community. But there's a lot of really passionate people that are working to provide better solutions.
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Mm-hmm. Okay. Okay. So let's go on the bright side. What excites you the most about newer approaches like the antigen testing, the enrichment technology, digital PCR? And does AI, is juicy AI playing a role here? I mean, my brain's kind of racing going, okay, well, if we took all these cases and fed all that into a...
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a model could start connecting dots that even we don't know how to connect? I think yes. And I think the first place that that starts is awareness. And actually we've started getting patient referrals because they put all their symptoms into AI and they're like, you might wanna consider vector-borne pathogens.
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And you might want to consider, you know, testing and Galaxy came up as providing testing solutions that are providing better answers. And so we're seeing it today in that people can take control of their own health and and really initiate some of their testing. We're working on a regular basis to figure out how do we make testing easier?
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How can we, you know, maybe empower telehealth or other places so that it's easy for a patient? Because that was my biggest problem is finding somebody that would go down the pathway with me. And we get frustrated because as much as we love a lot of the functional medicine providers that we work with, sometimes it can take months to get an appointment. And so where we can put more power into the patient's hands because...
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You know, I always, it's really intimidating when you have symptoms and you go into an expert, right? And then they say no, or they dismiss you or they gaslight you or they say it's not a big deal. And I always tell people, look, they're experts in whatever they're studying and whatever they do, but you're an expert in you.
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And you're an expert in the person that you're caring for. Because for me, it was being a caregiver. And if you know something is wrong, keep going until you find somebody that will go down that journey with you. And AI is one of those tools that you can use to just start,
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Figuring it out. What could it be? What are the exposures? Is this something that fits the symptoms that we're seeing? And so I'm excited by that, that opportunity. And then of course we're looking at it on the diagnostic side. How can we integrate information and, and testing from other types of other immune markers and cytokines and, and then direct detection? Because one of the problems that I think we've had with,
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with the biomarker approach is that that's a really useful thing. But again, with all these overlapping symptoms, if you can't correlate a biomarker expression panel with which pathogen is driving the bus, it's always going to be guessing. And the models might be able to figure it out, but the models are going to be a lot more powerful when you say, okay, this expression profile is guessing.
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aligned with a Babesia infection or aligned with a Bartonella infection or aligned with a, you know, or some combination of the two. And so that's going to allow the direct detection piece, which is what we're so focused on at Galaxy is going to enable a lot of those connections to become, you know,
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clearer. In this clip from episode number 435, our blank breaks down non-native EMFs and EMFs, the type of damage that happens to our DNA and how to spot good versus fake EMF protection products. This is invaluable information. Let's go back to EMFs because...
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you know, the earth gives off EMFs. Like EMFs are part of, electromagnetic frequency is a part of nature. But what we're talking about here are specifically non-native EMFs. And so I'd love to explain to the audience, people who are like obsessively optimizing their mitochondria, they're tracking their HRV, they're cold plunging at sunrise, they're doing all this crazy stuff, right?
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Let's talk about like what are non-native EMFs to someone who's a little bit new to this field or who's heard it and goes, oh, and my son said this to me once, like pretty soon you'll have us running around in tinfoil hats. And I said, well, as a matter of fact, I have this beanie. Yeah, I mean, it wouldn't be the worst thing in the world. I mean, maybe for their social lives. But so electromagnetic EMF stands for electromagnetic field.
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It's a form of radiation created by a combination of electricity and magnetism. And it was the discovery that electricity influences magnetism and magnetism influences electricity that gave birth, in my opinion, gave birth to the modern era. But that's where the studies of EMF came from, was this realization that these two key forces are fundamentally intertwined.
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and it exists on a spectrum, right? So there's a whole bunch of different types of EMF, and they're different based on their frequency, how quickly they vibrate. And not exactly in the center, but kind of in the middle of the electromagnetic spectrum is visible light. So all of everything we see from the sun, that is a form of EMF.
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And that, combined with the Earth's own magnetic field, is essentially, not entirely, but essentially the only form of EMF that all biological life was exposed to until the mid-19th century.
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Okay. Yeah. Okay. So we would have evolved with it, like all forms of nature would have evolved in that environment. Yes. And so there are forms of EMF with a lot more energy than visible light, and we call those ionizing.
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Because they have so much energy, they knock electrons out of your cells in your body. And that is very, very harmful, even very quickly in very low doses. And so we're talking about things like x-rays and gamma rays here. Right. So everyone knows you got to minimize that stuff. That's why the dental technician covers you in lead and leaves the room before they take the x-ray. Right. And so then- Needs you there. Yeah.
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And so then you have a whole bunch of EMFs with less energy than visible light. And because they have less energy than visible light, they can't ionize the cells in your body. And so that's why they're called non-ionizing. I think non-native is a little more consumer friendly, but technically they're called non-ionizing.
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And these are the forms that for centuries, scientists, doctors, engineers believed were biologically inert. That is, they were safe. And they include things like radio waves and microwaves. And so everything that runs on power...
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And everything that communicates wirelessly is a source of these non-ionizing EMFs or these non-native EMFs. And they're called non-native because all of them are human made. Right. So they all come from modern technology.
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When do you think we really started being exposed to, like was it with Edison's light bulb, the telephone, the invention of radio? Those are the things that really just started to bring these non-ionizing or non-ionizing into our space. Yeah, so it literally did start with the light bulb, except most people didn't have light bulbs when they invented the light bulb. So very, very few exposures.
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But there was the light bulb and then you needed a power grid so people could turn on lights in their own houses. And so then you get the power grid. And once there's a power grid in place, you can start doing all this other nifty stuff to run off that same power grid. And so that's when you get electrical appliances and power tools. And eventually...
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they realize that you can communicate wirelessly with this stuff. And so that's when you get radio and then television. Of course, militarily, you get radar. Then you later on get cordless phones and Wi-Fi and Bluetooth and near-field communication for Apple Pay and Google Pay. And so it did all start in the middle of the 19th century, but...
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the rate of growth has been massive and increasing. Yeah. And to underscore this point, oh, sorry. And so what rapid and increasing means is you and I have been exposed to orders of magnitude more EMF than our parents were. And our children will be exposed to orders of magnitude more than we have been and will have been.
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So these non-ionizing EMFs fundamentally affect our biology at a cellular level. Like this is... Yes. So, and this is what your dad and, sorry, I forget her name, the researcher... Reba Goodman, yeah. Yeah, really dove into. So mechanistically, let's, again, let's highlight for the audience how EMFs interact with the body. Like...
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Let's talk about things like oxidative stress, calcium channel disruption, mitochondrial dysfunction. And what's the strongest evidence we have right now? Because people are constantly- So yeah, everyone likes- Well, not likes, but-
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I understand that oxidative stress is a popular topic these days in the wellness community. And I understand why. And there is strong science on that point. But before we get to that, I want to talk about a set of studies that I personally find most compelling when trying to explain this to people. And this is a set of studies performed by doctors Henry Lai and Narendra Singh.
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Starting, I believe it was in the early 90s, although maybe it was in the late 80s. And what they, they were using cordless phones because cell phones weren't really a thing back then. And what they found is that even low levels of cordless phone radiation, even for short term exposure. So we're talking about 15 minutes led to significant increase in strand breaks of your DNA.
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And so strand breaks, that's when the two strands that form your DNA break. And you can either have a single strand break or a double strand break. If both strands of the DNA break, the cell will kill itself, which is actually a good outcome in this case. Because if only one strand breaks, the cell will try to recreate the broken strand. It won't repair the broken strand. It'll recreate the broken strand.
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And that is the, you know, a lot of times that works fine, but sometimes the cell messes up, especially the older that you get. And so that is the process by which mutations occur and mutations, everyone knows is a core mechanism behind the formation of disease.
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And one of the reasons I like to say, oh, and I should, I'm sorry, underscore, they performed a series of studies all on this same cause and effect. And this research has been replicated many, many times by other researchers in other laboratories around the world. And I'm sorry, I forgot one more point about this.
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This work shows, so I said, we're talking about short-term exposures, 15 minutes. They found that the increased rate of strand breaks persisted for hours following the exposure. So you had increased rates of strand breaks for that 15 minutes, but that increased rate of strand break continued for hours after the initial exposure. And this was when again? This was in the, it started, their initial work is in the early 90s.
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Wow. So, I mean, so this is back in the day when you could make a decision not to use a cordless phone, you know? Yes. Like, you could actually say, I can limit my exposure. At this stage of the game, even if I turn off the Wi-Fi in my house, I can see 10 other networks. Yeah. Easily. Like, easy. Probably more, because I'm downtown Toronto. Like, well, not downtown, but I'm, you know, I'm in an urban center.
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So let's bring it back to these more concrete physiological impacts. So you're talking about DNA damage right now, which is one of the hallmarks of aging, frankly. Oh, right. And so, yeah. So what does this mean? Also, if I may, it's not just a hallmark of aging. It's a mechanism by which disease forms. I said it before, but I just want to underscore that. That's a universally accepted mechanism for disease formation.
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But the other reason I really like using this set of science as an example is because not only does everyone understand implicitly what DNA is and how important it is, everyone also understands that DNA exists in almost every single cell of almost every single living thing on planet Earth.
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And so if there's a mechanism that impacts DNA in this way, you can understand why we are finding the health effects manifesting in every form of life in which we look.
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Let's talk about a couple of the most powerful free changes. Yeah. Yeah. So not carrying your phone in your pocket. Yeah. And before we continue on that, I just want to explain one thing about EMF. A characteristic of EMF is that the power of it diminishes exponentially with distance. It follows and this is all EMF radiation. It follows the inverse square law. And so what that means is if you have a cell phone and you're one inch away from it,
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and you then move it two inches away from your body, the power of your own exposure has dropped by 75%. And if you then go to four inches away, it's another 75%. And so when I say things like, don't carry it in your pocket, that's why. Because when it's right on your body, you are getting the maximum possible dose of this stuff. And moving it even a little bit away
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of this you are exposed to. And so that's why distance matters. And for the same reason, you shouldn't carry your phone in your pocket. You should certainly not hold it up to your head. So use speakerphone or wired headsets instead. Yeah.
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What about things like in the bedroom, right? Like how neurotic do people need to be about, like, I mean, there's people who've got- Neurosis isn't gonna help anybody. No, no, no. Yeah, that's gonna increase your oxygen. How diligent, let's use a different word. Let's use the word diligent. There are people who literally have breakers installed that cut the power to their bedrooms at night. And I would imagine in the electrosensitive world, hypersensitive world, that is a strategy they use. Yeah, because you can do that yourself
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with your own breaker panel. But if you're doing it and it's highly recommended, if you have high levels of electrical or magnetic fields in your bedroom, it is highly recommended to just turn off the breaker at night rather than trying to buy all this EMF protection. Yeah. Because it's, it's, it's in the walls, man. It's like, it's coming at you from all these different directions and mitigation and
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is very challenging, not only expensive, but actually challenging. So the solution is just to turn off to de-energize your bedroom. But the breakers you have in your breaker panel, by default, aren't really designed to be flipped on and off every day. And so that's why people buy special breakers that are more resilient for this type of behavior. And that is an incredibly effective technique. It turns out,
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because I had an assessment done at my house. It turns out I have shockingly low levels of EMF in my house. I'm sorry, in my bedroom. Well, actually in my whole house, but certainly in my bedroom. But one thing I did after the assessment is I had these pink Himalayan salt lamps because I just loved them. I have one, yeah. But they plug into the wall.
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And again, I didn't have really concerning levels of EMF, but she's told me like, you shouldn't have a plug-in wall lamp right next to your head. And I was like, oh yeah, you're right. Why didn't I think? So I ended up getting a, I have a low blue light lamp in my store.
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that's rechargeable. And so I can charge it in the kitchen and then just bring it to bed. And it also looks really cool. I really like it. It's kind of like Star Trek-y. But so I got rid of the lamps, the plug-in lamps in my bedroom and swapped them out for battery-operated rechargeables. But that's probably, you know, for most people,
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not having a TV in your bedroom, not bringing your cell phone to bed, those are going to be huge differences right off the bat. In this clip from episode number 447, Dr. Robin Rose goes deep on why long COVID has become more widely recognized, how COVID proliferates in the body, and what can be done to reduce the effects of that niggly little spike protein in the long term. There's two issues with the virus, okay? Okay.
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The virus can get into our human cells, just like how I explained the mRNA gets into the human cells. And it's not just the ACE2 receptor. That's like a teeny tiny part of the issue. Like I said, it can attach to over two dozen different host cell receptors in our body. OK, so once the virus gets a hold of you, right, we know studies have shown that you make trillions and trillions of spike protein.
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from just one exposure to SARS-CoV-2. Now that, now if the virus itself gets inside the cell two, then that cell now is a productive cell because the virus, just like the mRNA, takes over the DNA machinery and it's gonna just like start pumping out spikes. So it's a productive cell also. Right. So fine, so now you have
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We understand how the mRNA gets in there. The virus gets in there. These are both productive cells. Okay. But the virus also is a bacteriophage. This is also work done by Bronia and Cristoni's group in Italy. Okay. And the virus takes up residence inside the gut microbiome, particularly gram-positives.
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Phacobacterium prosmitcii and Dorea. I always mess up the other one. Dorea, I can never say the last part of it. Specifically, we found these, and these are major butyrate-producing species, okay? And you can see under the electron microscope when you read their paper that the bacteria are teeming with...
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hundreds if not thousands of SARS-CoV-2 inside of them. And once SARS-CoV-2 gets inside these bacteria, the bacteria start to then secrete these things called toxin-like peptides. And now you've layered a whole other thing
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Now we have a whole other layer of problems because the toxin-like peptides are very toxic to our body. And they are animal venom homologues, actually. So there's snake venom. No, no, sea snail venom. They're conotoxins. They're vasoactive. They're immune depleting. They affect the microvasculature like we've never seen. And they're extremely neurotoxic.
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Okay. And so they add another layer to all these issues with significant dysautonomia, with the microclotting. So it's like you have productive spike right out here in your cell systemically that can be driven by the virus or by the vaccine. Okay.
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And then you have the virus that takes hold of the gut microbiome and is also replicating, producing. And we now understand, and we have the testing to show, that the spike protein can also come from bacterial origin. So the bacteria are producing spike proteins.
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So when we thought that you just, that it could just be produced by our eukaryotic or human cells, it also can be produced by the prokaryotic or our bacterial cells that live with us. And so, and right. And so you have two, you have all these different origins now of spike protein production in the body. And yeah,
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Yeah, it's a, excuse my language, it's a shit show. It's a total shit show. So why are some people more affected than others? I mean, because not everybody falls apart with this, right? Like, I mean, some people... So I think a lot of things. I think that...
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people that already have a horribly broken microbiome, right? I mean, that's why we did so bad in the pandemic anyway, and we're such a sick population, because especially in America we do, most people walk around with significant dysbiasis, but people that were even worse off, right? And it was also, where is that person, right, in their health journey, right? Is it that last drop in the bucket, right? I think that,
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Most people were living right here, you know, and then getting the vaccine, getting the virus. It was sort of like, yeah, that last drop in the bucket. And then they basically manifested what they were maybe going to manifest even 10 years down the line. You know, who knows?
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So, so I think that that's a major problem. I think methylation, detoxification path, like all these things that how people, what people's pathways look like and how they're set up for that play a big, big role, right? So that they can detoxify, get rid of these, get rid of spike protein properly, all that stuff. Well, and what's interesting is something you said a minute ago, which was
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that even well patients come in and have a spike protein issue. So you could be asymptomatic and spike protein could silently be driving, accelerating aging under the surface. A hundred percent. I mean, there are already studies that I mean, nature, communication, nature. They've come out with studies already a good few years ago, just showing one exposure to COVID-19, like in a 19, 20, 21 year old and young people asymptomatic or a mild case have permanent brain changes.
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I mean, they have changes to their white matter, their gray matter. They have decreased cerebral blood flow on their, you know, on testing. Like they were able to show all this. This is not I'm not inventing this. This is all. Nobody wants to talk about it. I mean, you can even pull data right from the CDC. Right.
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pull data from whatever UK's CDC is, whatever Germany. It's not just here. It's all across the globe, right? And you can see the hundreds of millions of people living with this condition, which obviously is grossly underestimated because we know that it's not diagnosed or caught. Statistics show you right now that 30% of people that get COVID go on to have long COVID, right? Like symptomatically. Yeah.
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Yeah. Right. But then are but then, OK, but then what happens? Do they really get better? Are they better? And then where did where did that where does that fall off? Because then that doesn't translate into the fact that 18 million people they have, they say, living with long COVID. Right. Which is grossly underestimated also. And just to put that into context for the audience, like.
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That's how many people are living with a diagnosis of cancer. The good news is there's testing coming down the pipes are available. And there's treatments that you're very much involved in developing, which I think is amazing. Because there's been so little of that, right? In the silencing of dialogue, productive scientific dialogue at the very beginning, I think that...
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there was a huge delay in this kind of work and these discussions happening. So the good news is at least it's happening now. If you're listening to this and you or someone you know
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is experiencing long COVID and just isn't getting anywhere. Or just even like a condition that everyone's like, I don't know what this is. And you can temporarily relate it to when you got the vaccine or after you developed another case of COVID. You know, you need to get the right testing and right diagnostics so that we can understand like what's going on with you, you know?
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Absolutely. But then for everybody else who may be experiencing accelerated aging, but hasn't become symptomatic at this point, is there? What are the things? But even noticing certain little things. Yeah, but little things, right? Like, for example, I mean, you know, I haven't really spoken publicly about this, but I had an insanely difficult.
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over the top immune reaction to something at the beginning of the year. And there's a really good chance that it, you know, my immune system was kind of primed to get triggered by something that otherwise might not have done it. But what are things that people could be doing now
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I think it's medicinalsus.com. Yeah, you can, or just put into Google, like, medicinals9, it will come up the website. We'll find it. We'll find it. Yeah, right. So they sell, they do sell a pack where, you know, you can start doing, you know, and if you're sensitive, just, like, type, you know, desensitize yourself, you know, like, just start at much lower doses. Go low, go slow. You know, you could do, I would always do that first for, like, a good month, you know. And then I would start pulse dosing the Senolytics.
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You know, obviously get the binders on board too before you start this analytics so that the binders are all there ready to, you know, grab onto any spike protein. And is that any binder or there's specific binders? They have it. It's called, they have it. Everything is there. Everything's on that website? Yeah, you can get that. Okay.
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And then I would start doing like a lot of, you know, you know, and even maybe before I would say even before starting Medicinals 9 because a lot of people, you know, here in the West are a lot more sensitive. Right. Because we just don't have good gut microbiomes compared to, you know, European and Asians and they're used to the polyphenols and all that stuff because they just have it in their diet.
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And it's just cleaner. But I would do some gut work, you know, I would I mean, listen, I love lorazotide. That's not everyone can not everyone can get their hands on it. But I know there are some companies that, you know, have products like even like that have the lorazotide that are available. I'll list that.
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And there's a company that has both lorazotide on its own and lorazotide is part of gut healing. Right, right, exactly. And you can get like KPV, BPC-157, like all these things that are going to be really wonderful for helping the gut heal. I really like Thane, a biotic that's like a marvelous postbiotic that comes from...
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from healthy donor stool that has been, you know, treated and autoclaved. And so all the antigen, everything's killed. I will say start low and slow on that one. Yeah. I almost blew up my gut with that stuff. Yeah. I would do, you know, some of the, well, I don't always give that up front. I wait a little bit and I do some of the other work. I think the antibiotic, I think with the, once you get to that point, like even replacing bifidobacteria, that kind of foundational
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Well, you should do more of a spore former first, I think. So yeah, like I love Megaspore Biotic by Microbiome Labs, for example. Or Just Thrive is the other one you can get. People can get pretty easily. We'll put these links in the show notes. You can do that. And then, you know, just getting your diet right, you know, so that you can, you know, again, like get these good bacteria feeding on the right food because your diet is what really gives you those prebiotics, right? That's...
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And then everyone's, again, depending on what your unique microbiome looks like, depends on what you're feeding it. Because no one's alpha diversity, no one's beta is the same, no one's biome is the same. So you want to just be starting to eat clean, whole foods, non-processed foods, getting all the colors of the rainbow in.
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you know, like, I'm just trying to think what I do for, yeah, so I do all those things like zinc, you know, zinc, C and D are super important for gut healing, like getting that on board, it's going to be important as well. We talked about the probiotics, talked about diet, you know, just like sort of priming your gut, like so that you can start to take some of these things. So yeah, so
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I think taking the medicinals, taking then after getting that on board for a month and moving into like using the binders and the senolytics could be great because then you can liberate spike protein. And then, you know, as far as like, you know, we can put in the notes like, you know, things that you could do for your gut and getting rid of the phages as well. That's going to be really important.
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I hope you'll agree that this last quarter was filled with amazing guests throughout. So these are just a few moments that I really wanted to highlight. I hope this inspires you to return to any episodes you missed. Thank you again so much for being a listener and we'll see you next week. Hey folks, just a quick reminder that all of the information presented in this podcast is
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is for information purposes only. No medical advice, no diagnosing, no treatments suggested here. Before you try anything that you hear about or learn about here, make sure that you check with your medical provider.